Mould-infested flat forces family to flee every night over health fears

Mould-infested flat forces family to flee every night over health fears By Jennifer Hough Tuesday, March 29, 2011 A WOMAN and her three young children are being forced to leave their mould-infested home by night and sleep elsewhere due to health concerns linked to the condition of the flat. Slattery, 30, who lives in Dolphin House, Rialto, spoke to the Irish Examiner about the constant worry she lives with as her flat is covered in black mould which she feels could be damaging her children’s health. She and her three children, aged eight, five, and 10 months, have taken to sleeping in one bedroom at her mother’s house nearby. Ms Slattery is one of many residents of Dolphin’s House estate who are campaigning for better living conditions. They say that the daily health risk is a blatant violation of their right to adequate housing and called for a commitment from the Department of Environment to fund its regeneration. A study, published yesterday at a human rights monitoring hearing in Rialto, showed that 45% of adults and 42% of children living in the Dublin estate have trouble breathing easily and more than 90% are worried about their health. Residents reported diarrhoea and skin rashes, with some saying depression is also a consequence of living in a cold, damp smelly flat. "I have had the flat for eight and a half years now, and I’d say I was in it six months it when the problem first began. There was very bad dampness with white fluffy mould growing through the wall. The council came and treated it and re-wallpapered." Ms Slattery maintains, however, that in the past two years, it has "come back with a vengeance," but the council have not been back. "There is black mould around the windows even though they are PVC. "The walls in the bedroom are black, the wallpaper is peeling off, even my mattress is black and green and mouldy." Because of this, she says she doesn’t feel safe sleeping at the flat. "We stay in my sister’s room. She is away at college but she is back next month so I don’t know what we will do then. You are breathing it in all time, the kids were sick at the weekend — they were vomiting — I don’t know if it’s related but I am afraid it might be." As well as the mould, there is a rusty, leaking, waste pipe in the bathroom. "Other people’s waste runs down the pipe, it is just a trickle, but you can smell it. The pipe needs to be replaced, I have been waiting two years for that to happen. It is very stressful, my five-year-old asked me is she sick because of the mould. I was reared in these flats, and I just want somewhere safe and healthy to bring up my kids." A spokesperson for Dublin City Council said it is working with the residents to resolve maintenance issues. He said there is a monthly meeting between council representatives and the local maintenance group at which such matters and other issues are discussed. A spokesman for the Department of Environment said it has yet to receive a proposal from Dublin City Council with regards to Dolphin House. This story appeared in the printed version of the Irish Examiner Tuesday, March 29, 2011

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Health Risks

Health Risks: Moulds present a health hazard to humans because they release spores. These spores can cause allergic reactions and other respiratory problems especially in asthmatics. Eye irritation is also common. Some moulds produce mycotoxins which can result in severe neurological problems and even death after prolonged and untreated exposure. Symptoms of a mould allergy include watery, itchy eyes, chronic coughing , headaches or migraines, a difficulty breathing, inexplicable rashes, fatigue, sinus problems, nasal blockage and frequent sneezing. Anti-Mould Paint: This is a simple preventative method that involves using a specially formulated paint to deter the growth of moulds and mildews. This type of paint reduces condensation, insulating the wall and raising the temperature of the surface to reduce the amount of moisture and therefore mould in the home. This paint can be used for stippled or plain surfaces and greatly reduces the chance of mould and mildew growth. Remedial Steps: For smaller infestations, some less extreme measures may be taken. Exposing the mould growth to sunlight could reduce the problem. Similarly, improving ventilation in the room with mould can curb the mould's growth. Dehumidifiers and household cleansers may also have a positive effect on controlling the growth of the mould. However, some infestations require a more radical approach and professional removal. Mould Removal Dublin: Only trained professionals should attempt mould removal as close exposure to the spores can be dangerous. There are several methods for the successful removal of moulds in the home. • Dry ice blasting ' this method can be used on surfaces such as wood and cement. Soda and Media blasting are also efficient mould removal methods and are preferred to encapsulation which simply covers up the mould. • Vacuum - Wet vacuum cleaners can remove moulds from floors, carpets and other hard surfaces. This method should only be used if the mould is sufficiently wet otherwise spores may be exhausted into the indoor environment. • Damp wipe ' this form of Dublin mould treatment uses wiping or scrubbing moulds from non-porous surfaces with astringents. Fungicides should then be applied to prevent regrowth. • HEPA vacuum ' the high efficiency particulate air vacuum cleaners are used for clean-up sessions after others methods of removal have been used. All collected dust and debris should be stored in impervious bags to prevent unwanted release of spores. Tired of living with ugly and smelly mould colonies in your home? Concerned about the health of you and your loved ones? It might be time to consider professional Dublin mould removal. Need the help of specialists but concerned about the costs? We can help you find the best quality, affordable mould solutions at a price to suit your budget. Get a free, no-obligation quote from us today and wave goodbye to hazardous mould colonies.

Mucormycosis and Natural Disasters

Mucormycosis and Natural Disasters By Dr. Michelle Seidl, EMLab P&K Senior Analyst In 2012, Hurricane Sandy devastated parts of the Eastern seaboard and many areas of the Northern Caribbean. In 2011, a massive tornado wreaked havoc on Joplin, MO and nearby areas. In Southeast Asia in 2005, a tsunami left a destructive wake behind. These and other natural disasters are hotbeds for the occurrence and spread of various diseases. One example of an uncommon fungal disease that has been associated with various natural disasters is mucormycosis, also known more broadly as zygomycosis. Though the loss of life from this disease is very low when compared to the total loss of life caused by such horrific events, understanding the potential for this kind of infection could potentially save lives. Several people in Joplin, MO who were injured by the tornado developed a rare and potentially deadly fungal infection caused by a zygomycetous fungus. A recap of the May 22, 2011 event in relation to mucormycosis can be found on the CDC website. The physical and psychological damages the city of Joplin sustained were devastating. On June 3, 2011, a local physician notified the county and state health officials of two patients hospitalized with tornado-related injuries having suspected necrotizing (death of a specific area of tissue) fungal soft-tissue wound infections caused by a zygomycetous (mucoralean) fungus ((MMWR) July 29, 2011/60(29); 992.). The CDC and Missouri State Health Department immediately began active surveillance for such infections at hospitals and laboratories that were serving patients injured in the tornado. Within a week, eight patients with necrotizing fungal soft-tissue wound infections caused by mucoralean fungi were identified. About a month later, a total of 18 suspected cases of cutaneous (affecting the skin) mucormycosis had been identified, 13 of which were confirmed. A confirmed case here was defined as necrotizing soft tissue infection requiring antifungal treatment or surgical removal of the dead tissue, onset of the illness after May 22, 2011 and a positive fungal culture or histopathology and genetic sequencing consistent with a mucoralean fungus. No additional cases in that specific geographic area were reported after June 17, 2011. Of the 13 confirmed cases, 7 were female, 6 were male, all were white, and the age range was 13-76. Injuries sustained during the tornado included lacerations, fractures and blunt trauma. Two of the 13 patients had diabetes, none were immunocompromised, 10 required admission to an intensive care unit and 5 died. Specimens from all 13 patients yielded the mucoralean fungus: Apophysomyces trapeziformis.1 This fungus belongs to the fungal phylum Zygomycota, class Zygomycetes, subdivision Mucormycotina, order Mucorales, and classified in the family Mucoraceae. Since this tornado struck in late May during the growing season, spores could have been stirred up by the tornado, then dispersed to the victims through wounds from injuries or by ingestion or inhalation. All of those diagnosed with mucormycosis did have multiple injuries and secondary wound infections (Williams 2011). Following the Asian tsunami in 2004, a 56-year-old male survivor was diagnosed with an infection caused by another fungus in the same genus: Apophysomyces elegans.2 The survivor escaped broken bones but sustained traumatic wounds (Davis 2005). Despite cleaning and bandaging his wounds, he became feverish within 5 days. He was given broad spectrum antibiotics, the dead tissue cut away, and despite vigilant wound care, the fever remained and his condition worsened (Andreson, et al. 2005). This prompted a warning by doctors that survivors were at risk for this kind of fungal infection. In 1985, during one of the largest volcanic eruptions in recorded history, over 23,000 people died and 4,500 were seriously wounded. Of those wounded, 8 people were diagnosed with mucormycosis and 6 died. At the time, a plea was made for an early diagnosis, utilizing tissue sampling and microbiological studies, so that prompt and radical treatment could be instituted. This is especially pertinent in situations of natural disasters resulting in massive numbers of casualties and seriously injured survivors (Patiño, et al. 1991). Zygomycosis vs. Mucormycosis Zygomycosis is the most broadly encompassing term for a variety of diseases caused by fungi in the class Zygomycetes. Zygomycosis was originally described as a convenient and inclusive term for 2 clinically different diseases: mucormycosis caused by members of the order Mucorales and entomophthoramycosis caused by members of the order Entomophthorales (Kwong-Chun 2012). These two fungal orders comprise distinct lineages within the Zygomycetes (Hibbett et al. 2007; White et al. 2006). Most zygomycete infection cases are classified as mucormycosis. Of the rare clinical isolates identified as belonging to the Entomophthorales, most are caused by the genera Conidiobolus and Basidiobolus (Sugar 2007; Ribes, et al. 2000). Unfortunately, older literature describing cases of zygomycosis prior to molecular sequencing frequently did not identify the pathogen due to difficulty in culturing and due to lack of expertise needed to identify species using micromorphological methods (Iwen, et al. 2011). The general consensus is to have a combined approach which adopts both morphologic and molecular methods for species identification. In this article, and by definition, the term "mucormycosis" excludes members of the order Entomophthorales. In the laboratory, mucoralean fungi (e.g. the genus Mucor, see Figure 1), grow well on most standard fungal culture media such as Sabouraud dextrose agar. For a majority of the species associated with human disease, the growth is usually rapid with mycelial elements covering the entire plate within 2 to 3 days of incubation at 30°C. Unfortunately, the recovery of mucoralean fungi from tissue has been described as difficult, with negative results reported despite histological evidence of the presence of a zygomycete. Mucor spores and sporangia Figure 1: Mucor spores and sporangia. Copyright © 2013 EMLab P&K One reason for this inability to recover the fungus appears to be partly related to aggressive processing of the specimen that may damage the organism. A review by Roden, et al. (2005), described a clear increase in culture positivity over time with 71% of all cases since 2000 diagnosed on the basis of culture results. This improvement was suggested to be due to better training, a greater understanding of specimen processing, improved culture techniques, and increased access to sophisticated reference laboratories. The vegetative mycelium of all species in the Mucorales is composed of wide diameter, predominantly aseptate (nonseptate), colorless hyphae. The general growth characteristics useful for differentiation of members of this group include colony morphology, the presence of sporangiophores bearing multi-spored sporangia, and the presence or absence of rhizoids. Other methods helpful in identification include maximum temperature at which the isolate will grow and the ability of the organism to assimilate ethanol. Zygospore (sexual spore) formation would be another tool to use but is not always reproducible and can prove difficult. Mating studies require the maintenance of a library of tester strains and are often unrealistic. Fungal Genera Involved The most common genus involved in mucormycosis is Rhizopus (Fig. 2). Following is a list of genera that have been associated with this type of human infection: Rhizopus Mucor Rhizomucor Absidia (Lichtheimia) Apophysomyces Saksenaea Cunninghamella Cokeromyces Syncephalastrum Rhizopus spores and sporangium Figure 2: Rhizopus spores and sporangium. Copyright © 2013 EMLab P&K The Disease Mucormycosis is a very aggressive and severe infection, but is also very rare (Williams 2011). Organs and areas commonly affected include the sinuses, eyes, skin, brain and lungs. It may also affect the gastrointestinal tract, the skeletal system, the myocardium and endocardium, as well as the kidney (Walsh et al. 2012). It can also occur as a disseminated infection and play a role in allergic fungal sinusitis. The reference to cutaneous mucormycosis, translates to a disease caused by a member of the Mucorales and affecting the skin. Mucormycosis occurs primarily in people with immune disorders. It can occur, but is generally rare, in immunocompetent hosts. It is considered an opportunistic infection and often affects individuals with pre-existing conditions. Factors or conditions that are known to put humans at risk include: AIDS, diabetes mellitus (usually poorly controlled), lymphoma or leukemia, hematologic malignancy, neutropenia, organ transplants, sustained immunosuppressive therapy, long-term steroid use, metabolic acidosis, iron chelation therapy, broad-spectrum antibiotic use, injection drug use, protein or severe malnutrition and breakdown of the skin barrier such as trauma, surgical wounds, needle punctures or burns. The main risk factors for infection following a natural disaster are spore inhalation, spore ingestion and penetration through injuries that break the skin. Ibrahim et al. (2012), found that patients with elevated serum levels of available iron are susceptible to mucormycosis. These infections are highly angioinvasive (tendency to invade the walls of blood vessels), as the organism acquires iron from the host. This typically follows traumatic implantation or inhalation of the fungus. The disease is most common in the tropics, with cases reported from India, Australia, USA, Sri Lanka, Thailand, Central America and South America (Alvarez et al. 2010). Symptoms depend on the condition of the individual and the extent and location of the infection. If not diagnosed early, mucormycosis has an extremely high mortality rate (25% to 80%, averaging 40%). If properly diagnosed, the infection can be treated with antifungal agents (Davis 2005). At the time of this writing, according to the literature and the Mycotic Diseases Branch of the CDC, zero mucormycosis cases were reported as a result of Hurricane Sandy. Risk assessment is essential in post-disaster situations and the rapid implementation of control measures through re-establishment and improvement of primary healthcare delivery should be given high priority, especially in the absence of pre-disaster surveillance data (Kouadio, et al. 2012). As to whether or not we can say mucormycosis is definitively correlated with natural disasters, the jury is still out and more data will need to be gathered. For now, awareness is critical for prevention and treatment. If the disease is on the "radar screen" of attending physicians and workers following a disaster, the earlier clinical cases can be identified and treated, thereby avoiding this potentially deadly fungal infection from going undiagnosed. 1 Authors of this species: E. Álvarez, A. Stchigel, J. Cano, D. A. Sutton & J. Guarro. 2 Authors of this species: P.C. Misra, K.J. Srivastava & K. Lata. References: Alvarez, E, AM Stchigel, J Cano, DA Sutton, AW Fothergill, et al. 2010. Molecular phylogenetic diversity of the emerging, mucoralean fungus Apophysomyces: proposal of three new species. Rev. Iberoam. Micol. 27:80-89. Andresen, D, A Donaldson, L Choo, et al. 2005. Multifocal cutaneous mucormycosis complicating polymicrobial wound infections in a tsunami survivor from Sri Lanka. The Lancet 365(9462):876-878. doi:10.1016/S0140-6736(05)71046-1. Davis, K. 2005. Tsunami survivors risk deadly fungal infections. New Scientist 17:48. Hibbett, DS, M Binder, JF Bischoff, M Blackwell, PF Cannon, et al. 2007. A higher-level phylogenetic classification of the Fungi. Myc. Res. 111(5):509-547. Ibrahim, AS, Spellberg B, Walsh TJ, Kontoyiannis DP. 2012. Pathogenesis of mucormycosis. Clin Infect Dis. 54 Suppl 1:S16-22. doi: 10.1093/cid/cir865. Iwen, Peter C, I Thapa & D Bastola. 2011. Review of methods for the identification of Zygomycetes with an emphasis on molecular diagnostics. Lab medicine 42(5):260-266. DOI:10.1309/LMJ8Z0QPJ8BFVMZF Kouadio, IK, S Aljunid, T Kamigaki, K Hammad & H Oshitani. 2012. Infectious diseases following natural disasters: prevention and control measures. Expert Rev. Anti Infect. Ther 10(1): 95-104. Kwong-Chun, Kyung J. 2012. Taxonomy of Fungi Causing Mucormycosis and Entomophthoramycosis (Zygomycosis) and Nomenclature of the Disease: Molecular Mycologic Perspectives. Oxford University Press (Infectious Diseases Society of America). Clin Infect Dis. 54 (suppl 1): S8-S15. doi: 10.1093/cid/cir864 Patiño JF, Castro D, Valencia A, Morales P. 1991. Necrotizing soft tissue lesions after a volcanic cataclysm. World J Surg. Mar-Apr;15(2):240-7. Ribes, JA, CL Vanover-Sams & DJ Baker. 2000. Zygomycetes in human disease. Clin. Microbiol. Rev. 13(2): 236-301. Roden MM, Zaoutis TE, Buchanan WL, et al. 2005. Epidemiology and outcome of zygomycosis: A review of 929 reported cases. Clin Infect Dis. 41L634-653. Sugar, Alan M. 2007. Zygomycosis (Mucormycosis and Entomophthoromycosis). In: Carol A. Kauffman & G. L. Mandell (eds.). Atlas of fungal infections. Springer: Philadelphia. Walsh, Thomas J, BE Bloom & DP Kontoyiannis. 2012. Meeting the challenges of an emerging pathogen. The Henry Schueler 41 & 9 Foundation International Forum on Mucormycosis. Oxford University Press (Infectious Disease Society of America). Clin Infect Dis. 54 (suppl 1): S1-S4. doi: 10.1093/cid/cir862 White, Merlin M, Timothy Y James, Kerry O'Donnell, Matias J Cafaro, Yuuhiko Tanabe & Junta Sugiyam. 2006. Mycologia 98(6):872-884. Williams, T. 2011. 8 tornado victims stricken with rare fungal infection. 10 June 2011. The New York Times. http://seattletimes.com/html/health/2015289629_joplinfungus11.html The data and other information contained in this newsletter are provided for informational purposes only and should not be relied upon for any other purpose. EMLab P&K hereby disclaims any liability for any and all direct, indirect, punitive, incidental, special or consequential damages arising out of the use or interpretation of the data or other information contained in, or any actions taken or omitted in reliance upon, this newsletter. Images included in this newsletter are property of EMLab P&K, unless otherwise specified.

Toxic Mould

Stachybotrys chartarum, which is also known as the toxic mold. This fungus may produce spores, which are poisonous by inhalation. ‘The common places for mould to grow in houses is wallpaper, flooring, behind wall tiles and on window frames,’ explains Professor Richardson. He adds: ‘It can form in any poorly ventilated house, no matter how grand or ordinary, but it’s especially likely where there is moisture leaking.’ A leaking radiator is often a mould hotspot - you may not even be able to see that a radiator is leaking, but even a small leak can be enough to wet the back wall and the carpet beneath. Professor Roy Watling, an authority on fungi and formerly head of mycology at the Royal Botanic Garden, Edinburgh, says: 'When you walk around on the damp carpet, mould spores are released into the atmosphere, which you can then inhale. ‘Those most at risk of health problems caused by household moulds are children and babies, the elderly and those in poor health.’ Breathing in mould spores can have one of two effects - it can cause an infection, which usually strikes people with a weakened immune system. Mould can also cause allergic reactions, particularly asthma, as the immune system reacts to the spores when they make their way into the upper airways and sinuses. Symptoms of a mould problem include coughing, constant tiredness, eye and throat irritation, headaches, skin irritation or nausea.

Important Issues About Mould and How It Effects Your Children.

Important Issues About Mould and How It Effects Your Children. By: Jim Corkern Whether it is asthma, the flu, bronchitis, or any other type of illness, all parents are concerned with the long term effects of these conditions and what causes or aggravates them. Mould has somewhat taken centre stage in the list of things in your home that can cause adverse health effects for not only children and the elderly, but anyone else living in your home at the time, as well. Not only can mould cause the aggravation of allergies and respiratory conditions such as asthma and emphysema, but it can also be toxic and cause long term medical conditions that can even end in death in some severe cases. Due to their underdeveloped nature compared to adults, children are much more at risk to face the health effects of mould exposure than their adult counterparts. If your child has allergies, you probably keep an eye on their exposure to them on a daily basis, as well you should, but have you had your home tested for mould? If you have not, then you probably should. Your child's immune system is in development in the early years of its life and you should do what you can to make sure that the natural maturing process is not disturbed. Educate yourself about the different symptoms that can be caused from mould exposure, since many doctors are not particularly aware of the complex symptoms that can come about from the exposure. If you believe that your home has mould and your child is sick, relate this to your child's pediatrician. Infants are the most at risk, obviously, and if you suspect there is mold in your home anywhere, you should remove the child from the environment immediately and have tests done to determine if the mould is cosmetic or not. Hemorrhagic pneumonia can be caused by mould exposure in an infant and deaths have been reported. If your home has been flooded and your child's toys were exposed to flood waters, anything porous needs to be thrown away. This includes stuffed animals, pillows, and other bedding. These are no longer safe for your child to play with. Even mattresses need to be thrown out and replaced, because of the recent link between mould, crib mattresses, and Sudden Infant Death Syndrome.

How do moulds affect people?

How do molds affect people? Some people are sensitive to moulds. For these people, exposure to moulds can cause symptoms such as nasal stuffiness, eye irritation, wheezing, or skin irritation. Some people, such as those with serious allergies to molds, may have more severe reactions. Severe reactions may occur among workers exposed to large amounts of moulds in occupational settings, such as farmers working around moldy hay. Severe reactions may include fever and shortness of breath. Some people with chronic lung illnesses, such as obstructive lung disease, may develop mold infections in their lungs. In 2004 the Institute of Medicine (IOM) found there was sufficient evidence to link indoor exposure to mould with upper respiratory tract symptoms, cough, and wheeze in otherwise healthy people; with asthma symptoms in people with asthma; and with hypersensitivity pneumonitis in individuals susceptible to that immune-mediated condition. The IOM also found limited or suggestive evidence linking indoor mold exposure and respiratory illness in otherwise healthy children. In 2009, the World Health Organization issued additional guidance, the WHO Guidelines for Indoor Air Quality. Other recent studies have suggested a potential link of early mould exposure to development of asthma in some children, particularly among children who may be genetically susceptible to asthma development, and that selected interventions that improve housing conditions can reduce morbidity from asthma and respiratory allergies, but more research is needed in this regard.